adoK Family assigned · medium auto-curated

H37Rv Rv2202c · MTBC0 mtbc0_002338 · 324 aa · 2493210–2494184 (-) · RefSeq NP_216718.1

Annotation: from legacy to revised

Legacy (H37Rv / Mycobrowser)adenosine kinase
MTBC0 PGAP re-annotationcarbohydrate kinase family protein
Revised (this work)Carbohydrate kinase family protein. Pfam: PfkB (PF00294.30).

Auto-curated: this verdict and function were generated by rules from PGAP + Pfam + Foldseek and have not been hand-reviewed.

Curated reference (UniProt)

UniProt P9WID5 SwissProt · reviewed · Evidence at protein level
UniProt nameAdenosine kinase
EC (curated) EC 2.7.1.20
Curated functionCatalyzes the phosphorylation of adenosine to adenosine monophosphate (AMP). Can also catalyze the phosphorylation of the adenosine analog 2-methyladenosine (methyl-Ado) to methyl-AMP, the first step in the metabolism of this compound to an active form that displays antitubercular activity. Is not active on guanosine, inosine, deoxyadenosine, cytidine, uridine, or thymidine. Prefers dGTP and GTP to ATP as phosphate donors in vitro.

Functional vocabulary (eggNOG-mapper, orthology transfer)

COG category G Carbohydrate transport and metabolism
Preferred nameadoK
eggNOG descriptionRibokinase
Orthologous groupCOG0524
EC number EC 2.7.1.20
KEGG orthology K00856
KEGG pathways map00230, map01100
Gene Ontology (109) GO:0000166, GO:0000287, GO:0001882, GO:0001883, GO:0003674, GO:0003824, GO:0004001, GO:0005488, GO:0005524, GO:0005525, GO:0005575, GO:0005622 +97 more

Orthology-based transfer (eggNOG 5.0.2, diamond). EC/KO/GO/CAZy are computed annotations, not manual curation; cross-check against the primary literature before treating a specific reaction as established.

Conservation & selection (intra-MTBC, 145 209 strains)

pN/pS 0.086 · strong purifying
Polymorphic sites (≥ 0.1% of strains) 8 synonymous, 2 missense, 0 nonsense, 0 frameshift

pN/pS from segregating SNPs (singletons removed) normalised by possible sites. Low pN/pS = purifying selection (a strong signal that a "hypothetical" is a real, constrained gene). A high pN/pS is ambiguous: relaxed constraint or positive selection (drug resistance, antigenic variation) inflate it; e.g. rpoB/katG/pncA score high here for resistance, not loss of function. A clonal disruption (one allele over a clade) suggests lineage pseudogenisation; a convergent one (many independent alleles) is typical of resistance loss-of-function.

Domains (Pfam, hmmscan --cut_ga)

PfamAccessioni-EvalueResiduesDescription
PfkBPF00294.30 3.4e-5215–295 pfkB family carbohydrate kinase

Functional interaction network (STRING v12, guilt-by-association)

Closest characterised functional partner: Rv2203 (membrane protein), high confidence from genomic context alone (score 758 excluding text-mining).

PartnerProductScoreNo text-miningChannels (≥400)
Rv2584c apt adenine phosphoribosyltransferase 963 948 database:900
Rv0777 purB adenylosuccinate lyase PurB 945 922 database:900
Rv0733 adk adenylate kinase 974 920 database:900 textmining:691
Rv3393 iunH nucleoside hydrolase 925 915 database:900
Rv3313c add adenosine deaminase 991 914 database:900 textmining:901
Rv3307 deoD purine nucleoside phosphorylase 938 914 database:900
Rv0805 cpdA 3',5'-cyclic adenosine monophosphate phosphodiesterase CpdA 906 906 database:900
Rv2203 membrane protein 758 758 ctx neighborhood:757
Rv1603 hisA 1-(5-phosphoribosyl)-5-((5-phosphoribosylamino)methylideneamino)imidazole-4-carboxamide isomerase 713 702 coexpression:692
Rv0498 hyp hypothetical protein 612 560 coexpression:535
Rv1752 hyp hypothetical protein 519 458 coexpression:427
Rv3248c sahH adenosylhomocysteinase 745 452 textmining:554
Rv1392 metK S-adenosylmethionine synthetase 512 450
Rv0357c purA adenylosuccinate synthetase 600 447
Rv1416 ribH 6,7-dimethyl-8-ribityllumazine synthase 513 447 coexpression:429

STRING combines evidence channels (neighborhood, fusion, cooccurrence, coexpression, experimental, database, text-mining) into a 0–1000 score. The ctx badge marks edges carried by the genomic-context channels (conserved neighborhood, fusion, phylogenetic co-occurrence), which are independent of orthology and structure and the strongest signal for an unknown gene. The no text-mining column recomputes the score from data alone, so a link that does not depend on the literature is visible. Association is a function hypothesis, not proof: corroborate with the operon context and the primary literature before assigning a function.

Evidence

  • Legacy H37Rv annotation: adenosine kinase
  • MTBC0 PGAP product: carbohydrate kinase family protein
  • Pfam (hmmscan --cut_ga): PfkB PF00294.30 (E=3e-52)
  • (auto-curated by rules from PGAP + Pfam + Foldseek; not hand-reviewed)

Sources

  • Ancestral sequence & coordinates: Harrison LB et al. (2024), An imputed ancestral reference genome for the MTBC, doi:10.1101/2023.09.07.556366
  • Product annotation: NCBI PGAP on MTBC0; legacy from H37Rv NC_000962.3 (RefSeq NP_216718.1)
  • Domains: Pfam-A via hmmscan --cut_ga — PfkB (PF00294.30)
  • Sequence-level signal: ESM Atlas (EvolutionaryScale × BioHub) — exploratory
  • Controlled vocabulary: eggNOG-mapper 2.1.12 (Cantalapiedra et al. 2021, doi:10.1093/molbev/msab293), eggNOG 5.0 DB (Huerta-Cepas et al. 2019) — OG COG0524
  • Curated reference: UniProt P9WID5 (SwissProt, reviewed; Evidence at protein level)
  • Intra-MTBC selection: pN/pS and disruption from SPDI variants of 145 209 MTBC strains (this work, local collection vs H37Rv NC_000962.3)
  • Interaction network: STRING v12.0 (Szklarczyk et al. 2023, doi:10.1093/nar/gkac1000), taxon 83332, CC-BY 4.0 — 43 functional partner(s); context anchor Rv2203
  • Primary literature: none located yet; annotation rests on the domain/homology sources above.

Ancestral MTBC0 protein sequence

>mtbc0_002338|Rv2202c|adoK
MTIAVTGSIATDHLMRFPGRFSEQLLPEHLHKVSLSFLVDDLVMHRGGVAGNMAFAIGVLGGEVALVGAAGADFADYRDWLKARGVNCDHVLISETAHTARFTCTTDVDMAQIASFYPGAMSEARNIKLADVVSAIGKPELVIIGANDPEAMFLHTEECRKLGLAFAADPSQQLARLSGEEIRRLVNGAAYLFTNDYEWDLLLSKTGWSEADVMAQIDLRVTTLGPKGVDLVEPDGTTIHVGVVPETSQTDPTGVGDAFRAGFLTGRSAGLGLERSAQLGSLVAVLVLESTGTQEWQWDYEAAASRLAGAYGEHAAAEIVAVLA