rsdA Resolved · high auto-curated

H37Rv Rv3413c · MTBC0 mtbc0_003627 · 299 aa · 3857585–3858484 (-) · RefSeq NP_217930.1

Annotation: from legacy to revised

Legacy (H37Rv / Mycobrowser)anti-sigma-D factor RsdA
MTBC0 PGAP re-annotationanti-sigma D factor RsdA
Revised (this work)Anti-sigma D factor RsdA. Pfam: RsdA_SigD_bd (PF16751.11).

Auto-curated: this verdict and function were generated by rules from PGAP + Pfam + Foldseek and have not been hand-reviewed.

Curated reference (UniProt)

UniProt P9WJ71 SwissProt · reviewed · Evidence at protein level
UniProt nameAnti-sigma-D factor RsdA
Curated functionAn anti-sigma factor for extracytoplasmic function (ECF) sigma factor SigD. ECF sigma factors are held in an inactive form by an anti-sigma factor until released by regulated intramembrane proteolysis (RIP). RIP occurs when an extracytoplasmic signal triggers a concerted proteolytic cascade to transmit information and elicit cellular responses. The membrane-spanning regulatory substrate protein is first cut extracytoplasmically (site-1 protease, S1P), then within the membrane itself (site-2 protease, S2P), while cytoplasmic proteases finish degrading the regulatory protein, liberating the sigm.

Functional vocabulary (eggNOG-mapper, orthology transfer)

COG category S Function unknown
Preferred namersdA
eggNOG descriptionAnti-sigma-D factor RsdA to sigma factor binding region
Orthologous group2DVAJ
Gene Ontology (2) GO:0005575, GO:0005576

Orthology-based transfer (eggNOG 5.0.2, diamond). EC/KO/GO/CAZy are computed annotations, not manual curation; cross-check against the primary literature before treating a specific reaction as established.

Conservation & selection (intra-MTBC, 145 209 strains)

pN/pS n/a
Polymorphic sites (≥ 0.1% of strains) 0 synonymous, 2 missense, 0 nonsense, 1 frameshift
Disruption 1 distinct premature-stop/frameshift site(s); most common in 0.38% of strains (558) · clonal

pN/pS from segregating SNPs (singletons removed) normalised by possible sites. Low pN/pS = purifying selection (a strong signal that a "hypothetical" is a real, constrained gene). A high pN/pS is ambiguous: relaxed constraint or positive selection (drug resistance, antigenic variation) inflate it; e.g. rpoB/katG/pncA score high here for resistance, not loss of function. A clonal disruption (one allele over a clade) suggests lineage pseudogenisation; a convergent one (many independent alleles) is typical of resistance loss-of-function.

Domains (Pfam, hmmscan --cut_ga)

PfamAccessioni-EvalueResiduesDescription
RsdA_SigD_bdPF16751.11 7.0e-1714–58 Anti-sigma-D factor RsdA to sigma factor binding region

Functional interaction network (STRING v12, guilt-by-association)

Closest characterised functional partner: sigD (ECF RNA polymerase sigma factor SigD), high confidence from genomic context alone (score 1000 excluding text-mining).

PartnerProductScoreNo text-miningChannels (≥400)
Rv3414c sigD ECF RNA polymerase sigma factor SigD 999 1000 ctx neighborhood:882 cooccurence:488 experimental:999
Rv3415c hyp hypothetical protein 868 869 ctx neighborhood:767 cooccurence:459
Rv0210 hyp hypothetical protein 746 746 ctx cooccurence:746
Rv0556 transmembrane protein 714 714 ctx cooccurence:714
Rv2360c hyp hypothetical protein 712 712 ctx cooccurence:712
Rv0996 transmembrane protein 711 711 ctx cooccurence:711
Rv0481c hyp hypothetical protein 699 699 ctx cooccurence:699
Rv2138 lppL lipoprotein LppL 694 695 ctx cooccurence:654
Rv3071 hyp hypothetical protein 689 689 ctx cooccurence:689
Rv3438 hyp hypothetical protein 681 682 ctx cooccurence:672
Rv2474c hyp hypothetical protein 668 668 ctx cooccurence:668
Rv2732c transmembrane protein 667 667 ctx cooccurence:667
Rv3217c integral membrane protein 665 665 ctx cooccurence:665
Rv0431 tuberculin-like peptide 663 663 ctx cooccurence:663
Rv3212 hyp hypothetical protein 661 661 ctx cooccurence:660

STRING combines evidence channels (neighborhood, fusion, cooccurrence, coexpression, experimental, database, text-mining) into a 0–1000 score. The ctx badge marks edges carried by the genomic-context channels (conserved neighborhood, fusion, phylogenetic co-occurrence), which are independent of orthology and structure and the strongest signal for an unknown gene. The no text-mining column recomputes the score from data alone, so a link that does not depend on the literature is visible. Association is a function hypothesis, not proof: corroborate with the operon context and the primary literature before assigning a function.

Evidence

  • Legacy H37Rv annotation: anti-sigma-D factor RsdA
  • MTBC0 PGAP product: anti-sigma D factor RsdA
  • Pfam (hmmscan --cut_ga): RsdA_SigD_bd PF16751.11 (E=7e-17)
  • (auto-curated by rules from PGAP + Pfam + Foldseek; not hand-reviewed)

Sources

  • Ancestral sequence & coordinates: Harrison LB et al. (2024), An imputed ancestral reference genome for the MTBC, doi:10.1101/2023.09.07.556366
  • Product annotation: NCBI PGAP on MTBC0; legacy from H37Rv NC_000962.3 (RefSeq NP_217930.1)
  • Domains: Pfam-A via hmmscan --cut_ga — RsdA_SigD_bd (PF16751.11)
  • Sequence-level signal: ESM Atlas (EvolutionaryScale × BioHub) — exploratory
  • Controlled vocabulary: eggNOG-mapper 2.1.12 (Cantalapiedra et al. 2021, doi:10.1093/molbev/msab293), eggNOG 5.0 DB (Huerta-Cepas et al. 2019) — OG 2DVAJ
  • Curated reference: UniProt P9WJ71 (SwissProt, reviewed; Evidence at protein level)
  • Intra-MTBC selection: pN/pS and disruption from SPDI variants of 145 209 MTBC strains (this work, local collection vs H37Rv NC_000962.3)
  • Interaction network: STRING v12.0 (Szklarczyk et al. 2023, doi:10.1093/nar/gkac1000), taxon 83332, CC-BY 4.0 — 95 functional partner(s); context anchor sigD
  • Primary literature: none located yet; annotation rests on the domain/homology sources above.

Ancestral MTBC0 protein sequence

>mtbc0_003627|Rv3413c|rsdA
MREFGNPLGDRPPLDELARTDLLLDALAEREEVDFADPRDDALAALLGQWRDDLRWPPASALVSQDEAVAALRAGVAQRRRARRSLAAVGSVAAALLVLSGFGAVVADARPGDLLYGLHAMMFNRSRVSDDQIVLSAKANLAKVEQMIAQGQWAEAQDELAEVSSTVQAVTDGSRRQDLINEVNLLNTKVETRDPNATLRPGSPSNPAAPGSVGNSWTPLAPVVEPPTPPTPASAAEPSMSAGVSESPMPNSTSTVAASPSTPSSKPEPGSIDPSLEPADEATNPAGQPAPETPVSPTH