echA13 Resolved · high auto-curated

H37Rv Rv1935c · MTBC0 mtbc0_002049 · 318 aa · 2205319–2206275 (-) · RefSeq NP_216451.1

Annotation: from legacy to revised

Legacy (H37Rv / Mycobrowser)enoyl-CoA hydratase EchA13
MTBC0 PGAP re-annotationenoyl-CoA hydratase
Revised (this work)Enoyl-CoA hydratase. Pfam: ECH_2 (PF16113.11), ECH_1 (PF00378.26).

Auto-curated: this verdict and function were generated by rules from PGAP + Pfam + Foldseek and have not been hand-reviewed.

Curated reference (UniProt)

UniProt P95279 SwissProt · reviewed · Evidence at transcript level
UniProt namePutative enoyl-CoA hydratase EchA13

UniProt still lists this protein as Putative enoyl-CoA hydratase EchA13; the revised annotation above is ahead of the current UniProt record.

Functional vocabulary (eggNOG-mapper, orthology transfer)

COG category I Lipid transport and metabolism
Preferred nameechA13
eggNOG descriptionEnoyl-CoA hydratase
Orthologous groupCOG1024
EC number EC 4.2.1.17
KEGG orthology K01692
KEGG pathways map00071, map00280, map00281, map00310, map00360, map00362, map00380, map00410, map00627, map00640, map00650, map00903, map00930, map01100, map01110, map01120, map01130, map01212
KEGG modules M00032, M00087

Orthology-based transfer (eggNOG 5.0.2, diamond). EC/KO/GO/CAZy are computed annotations, not manual curation; cross-check against the primary literature before treating a specific reaction as established.

Conservation & selection (intra-MTBC, 145 209 strains)

pN/pS 0.219 · purifying
Polymorphic sites (≥ 0.1% of strains) 6 synonymous, 3 missense, 1 nonsense, 0 frameshift
Disruption 1 distinct premature-stop/frameshift site(s); most common in 0.24% of strains (355) · clonal

pN/pS from segregating SNPs (singletons removed) normalised by possible sites. Low pN/pS = purifying selection (a strong signal that a "hypothetical" is a real, constrained gene). A high pN/pS is ambiguous: relaxed constraint or positive selection (drug resistance, antigenic variation) inflate it; e.g. rpoB/katG/pncA score high here for resistance, not loss of function. A clonal disruption (one allele over a clade) suggests lineage pseudogenisation; a convergent one (many independent alleles) is typical of resistance loss-of-function.

Domains (Pfam, hmmscan --cut_ga)

PfamAccessioni-EvalueResiduesDescription
ECH_2PF16113.11 3.2e-1538–244 Enoyl-CoA hydratase/isomerase
ECH_1PF00378.26 7.5e-23110–262 Enoyl-CoA hydratase/isomerase

Functional interaction network (STRING v12, guilt-by-association)

Closest characterised functional partner: fadE17 (acyl-CoA dehydrogenase FadE17), high confidence from genomic context alone (score 991 excluding text-mining).

PartnerProductScoreNo text-miningChannels (≥400)
Rv1934c fadE17 acyl-CoA dehydrogenase FadE17 993 991 ctx neighborhood:869 coexpression:900
Rv1933c fadE18 acyl-CoA dehydrogenase FadE18 934 888 ctx neighborhood:747 textmining:439
Rv1936 monooxygenase 908 882 ctx neighborhood:492 coexpression:731
Rv2500c fadE19 acyl-CoA dehydrogenase FadE19 851 845 database:750
Rv3140 fadE23 acyl-CoA dehydrogenase FadE23 859 844 database:750
Rv0975c fadE13 acyl-CoA dehydrogenase FadE13 850 844 database:750
Rv0131c fadE1 acyl-CoA dehydrogenase FadE1 850 844 database:750
Rv0400c fadE7 acyl-CoA dehydrogenase FadE7 849 844 database:750
Rv0231 fadE4 acyl-CoA dehydrogenase FadE4 849 844 database:750
Rv0154c fadE2 acyl-CoA dehydrogenase FadE2 849 844 database:750
Rv1966 mce3A Mce family protein Mce3A 839 810 coexpression:810
Rv1965 yrbE3B integral membrane protein 805 805 coexpression:805
Rv2524c fas fatty acid synthase 816 789 coexpression:645
Rv3540c ltp2 lipid transfer protein 797 783 database:447
Rv0468 fadB2 3-hydroxybutyryl-CoA dehydrogenase 791 783 database:650

STRING combines evidence channels (neighborhood, fusion, cooccurrence, coexpression, experimental, database, text-mining) into a 0–1000 score. The ctx badge marks edges carried by the genomic-context channels (conserved neighborhood, fusion, phylogenetic co-occurrence), which are independent of orthology and structure and the strongest signal for an unknown gene. The no text-mining column recomputes the score from data alone, so a link that does not depend on the literature is visible. Association is a function hypothesis, not proof: corroborate with the operon context and the primary literature before assigning a function.

Evidence

  • Legacy H37Rv annotation: enoyl-CoA hydratase EchA13
  • MTBC0 PGAP product: enoyl-CoA hydratase
  • Pfam (hmmscan --cut_ga): ECH_2 PF16113.11 (E=3e-15), ECH_1 PF00378.26 (E=7e-23)
  • (auto-curated by rules from PGAP + Pfam + Foldseek; not hand-reviewed)

Sources

  • Ancestral sequence & coordinates: Harrison LB et al. (2024), An imputed ancestral reference genome for the MTBC, doi:10.1101/2023.09.07.556366
  • Product annotation: NCBI PGAP on MTBC0; legacy from H37Rv NC_000962.3 (RefSeq NP_216451.1)
  • Domains: Pfam-A via hmmscan --cut_ga — ECH_2 (PF16113.11), ECH_1 (PF00378.26)
  • Sequence-level signal: ESM Atlas (EvolutionaryScale × BioHub) — exploratory
  • Controlled vocabulary: eggNOG-mapper 2.1.12 (Cantalapiedra et al. 2021, doi:10.1093/molbev/msab293), eggNOG 5.0 DB (Huerta-Cepas et al. 2019) — OG COG1024
  • Curated reference: UniProt P95279 (SwissProt, reviewed; Evidence at transcript level)
  • Intra-MTBC selection: pN/pS and disruption from SPDI variants of 145 209 MTBC strains (this work, local collection vs H37Rv NC_000962.3)
  • Interaction network: STRING v12.0 (Szklarczyk et al. 2023, doi:10.1093/nar/gkac1000), taxon 83332, CC-BY 4.0 — 150 functional partner(s); context anchor fadE17
  • Primary literature: none located yet; annotation rests on the domain/homology sources above.

Ancestral MTBC0 protein sequence

>mtbc0_002049|Rv1935c|echA13
MFVGRVGPVDRRSDGERSRRPREFEYIRYETIDDGRIAAITLDRPKQRNAQTRGMLVELGAAFELAEADDTVRVVILRAAGPAFSAGHDLGSADDIRERSPGPDQHPSYRCNGATFGGVESRNRQEWHYYFENTKRWRNLRKITIAQVHGAVLSAGLMLAWCCDLIVASEDTVFADVVGTRLGMCGVEYFGHPWEFGPRKTKELLLTGDCIGADEAHALGMVSKVFPADELATSTIEFARRIAKVPTMAALLIKESVNQTVDAMGFSAALDGCFKIHQLNHAHWGEVTGGKLSYGTVEYGLEDWRAAPQIRPAIKQRP