Rv0894 Family assigned · medium auto-curated

H37Rv Rv0894 · MTBC0 mtbc0_000948 · 393 aa · 999738–1000919 (+) · RefSeq NP_215409.1

Annotation: from legacy to revised

Legacy (H37Rv / Mycobrowser)transcriptional regulator
MTBC0 PGAP re-annotationhypothetical protein
Revised (this work)Contains HTH_77 (PF25872.1) domain(s); putative function inferred from the domain architecture.

Auto-curated: this verdict and function were generated by rules from PGAP + Pfam + Foldseek and have not been hand-reviewed.

Curated reference (UniProt)

UniProt P9WKP9 SwissProt · reviewed · Predicted
UniProt nameUncharacterized protein Rv0894

UniProt still lists this protein as Uncharacterized protein Rv0894; the revised annotation above is ahead of the current UniProt record.

Functional vocabulary (eggNOG-mapper, orthology transfer)

COG category K Transcription
eggNOG descriptioninvolved in signal transduction (via phosphorylation) involved in transcriptional regulatory mechanism and in the regulation of secondary metabolites catalytic activity ATP a protein ADP a phosphoprotein
Orthologous groupCOG3903
Gene Ontology (10) GO:0005575, GO:0005622, GO:0005623, GO:0005737, GO:0005829, GO:0008150, GO:0040007, GO:0044424, GO:0044444, GO:0044464

Orthology-based transfer (eggNOG 5.0.2, diamond). EC/KO/GO/CAZy are computed annotations, not manual curation; cross-check against the primary literature before treating a specific reaction as established.

Conservation & selection (intra-MTBC, 145 209 strains) pseudogene candidate

pN/pS 0.278 · purifying
Polymorphic sites (≥ 0.1% of strains) 5 synonymous, 4 missense, 0 nonsense, 1 frameshift
Disruption 1 distinct premature-stop/frameshift site(s); most common in 5.92% of strains (8597) · clonal

pN/pS from segregating SNPs (singletons removed) normalised by possible sites. Low pN/pS = purifying selection (a strong signal that a "hypothetical" is a real, constrained gene). A high pN/pS is ambiguous: relaxed constraint or positive selection (drug resistance, antigenic variation) inflate it; e.g. rpoB/katG/pncA score high here for resistance, not loss of function. A clonal disruption (one allele over a clade) suggests lineage pseudogenisation; a convergent one (many independent alleles) is typical of resistance loss-of-function.

Domains (Pfam, hmmscan --cut_ga)

PfamAccessioni-EvalueResiduesDescription
HTH_77PF25872.1 7.3e-10284–360 Winged helix-turn-helix domain

Functional interaction network (STRING v12, guilt-by-association)

Closest characterised functional partner: moaR1 (transcriptional regulator MoaR), high confidence from genomic context alone (score 989 excluding text-mining).

PartnerProductScoreNo text-miningChannels (≥400)
Rv3124 moaR1 transcriptional regulator MoaR 996 989 ctx fusion:899 cooccurence:489 coexpression:802 textmining:663
Rv1267c embR transcriptional regulator EmbR 968 967 ctx fusion:774 coexpression:800
Rv0891c hyp hypothetical protein 965 965 ctx fusion:756 coexpression:801
Rv0895 diacyglycerol O-acyltransferase 908 908 ctx neighborhood:559 coexpression:801
Rv1359 transcriptional regulator 893 894 coexpression:860
Rv3167c TetR family transcriptional regulator 861 861 coexpression:834
Rv0602c tcrA two component DNA binding transcriptional regulator TcrA 850 845 coexpression:845
Rv3263 DNA methylase 841 841 coexpression:841
Rv0691c mftR mycofactocin biosynthesis transcriptional regulator MftR 841 841 coexpression:841
Rv3840 transcriptional regulator 834 834 coexpression:834
Rv1675c cmr HTH-type transcriptional regulator Cmr 834 834 coexpression:834
Rv1189 sigI ECF RNA polymerase sigma factor SigI 833 833 coexpression:791
Rv3736 AraC/XylS family transcriptional regulator 833 833 coexpression:833
Rv1931c transcriptional regulator 828 829 coexpression:801
Rv1725c hyp hypothetical protein 826 826 coexpression:800

STRING combines evidence channels (neighborhood, fusion, cooccurrence, coexpression, experimental, database, text-mining) into a 0–1000 score. The ctx badge marks edges carried by the genomic-context channels (conserved neighborhood, fusion, phylogenetic co-occurrence), which are independent of orthology and structure and the strongest signal for an unknown gene. The no text-mining column recomputes the score from data alone, so a link that does not depend on the literature is visible. Association is a function hypothesis, not proof: corroborate with the operon context and the primary literature before assigning a function.

Evidence

  • Legacy H37Rv annotation: transcriptional regulator
  • MTBC0 PGAP product: hypothetical protein
  • Pfam (hmmscan --cut_ga): HTH_77 PF25872.1 (E=7e-10)
  • (auto-curated by rules from PGAP + Pfam + Foldseek; not hand-reviewed)

Sources

  • Ancestral sequence & coordinates: Harrison LB et al. (2024), An imputed ancestral reference genome for the MTBC, doi:10.1101/2023.09.07.556366
  • Product annotation: NCBI PGAP on MTBC0; legacy from H37Rv NC_000962.3 (RefSeq NP_215409.1)
  • Domains: Pfam-A via hmmscan --cut_ga — HTH_77 (PF25872.1)
  • Sequence-level signal: ESM Atlas (EvolutionaryScale × BioHub) — exploratory
  • Controlled vocabulary: eggNOG-mapper 2.1.12 (Cantalapiedra et al. 2021, doi:10.1093/molbev/msab293), eggNOG 5.0 DB (Huerta-Cepas et al. 2019) — OG COG3903
  • Curated reference: UniProt P9WKP9 (SwissProt, reviewed; Predicted)
  • Intra-MTBC selection: pN/pS and disruption from SPDI variants of 145 209 MTBC strains (this work, local collection vs H37Rv NC_000962.3)
  • Interaction network: STRING v12.0 (Szklarczyk et al. 2023, doi:10.1093/nar/gkac1000), taxon 83332, CC-BY 4.0 — 79 functional partner(s); context anchor moaR1
  • Primary literature: none located yet; annotation rests on the domain/homology sources above.

Ancestral MTBC0 protein sequence

>mtbc0_000948|Rv0894|
MPSRATVQEFSDSYPFCHNGFRPIMMPKIVSVQHSTRRHLTSFVGRKAELNDVRRLLSDKRLVTLTGPDGMGKSRLALQIGAQIAHEFTYGRWDCDLATVTDRDCVSISMLNALGLPVQPGLSAIDTLVGVINDARVLLVLDHCEHLLDACAAIIDSLLRSCPRLTILTTSTEAIGLAGELTWRVPPLSLTNDAIELFVDRARRVRSDFAINADTAVTVGEICRRLDGVPLAIELAAARTDTLSPVEILAGLNDRFRLVAGAAGNAVRPEQTLCATVQWSHALLSGPERALLHRLAVFAGGFDLDGAQAVGANDEDFEGYQTLGRFAELVDKAFVVVENNRGRAGYRLLYSVRQYALEKLSESGEADAVLARYRKHLKQPNQVVRAGSGGVRY