Rv0897c Resolved · high auto-curated

H37Rv Rv0897c · MTBC0 mtbc0_000951 · 535 aa · 1004022–1005629 (-) · RefSeq NP_215412.1

Annotation: from legacy to revised

Legacy (H37Rv / Mycobrowser)oxidoreductase
MTBC0 PGAP re-annotationNAD(P)/FAD-dependent oxidoreductase
Revised (this work)NAD(P)/FAD-dependent oxidoreductase. Pfam: FAD_binding_3 (PF01494.26), Thi4 (PF01946.24), FAD_oxidored (PF12831.14), GIDA (PF01134.29), FAD_binding_2 (PF00890.31), NAD_binding_8 (PF13450.13).

Auto-curated: this verdict and function were generated by rules from PGAP + Pfam + Foldseek and have not been hand-reviewed.

Curated reference (UniProt)

UniProt P9WKP7 SwissProt · reviewed · Evidence at protein level
UniProt nameUncharacterized protein Rv0897c

UniProt still lists this protein as Uncharacterized protein Rv0897c; the revised annotation above is ahead of the current UniProt record.

Functional vocabulary (eggNOG-mapper, orthology transfer)

COG category Q Secondary metabolites biosynthesis, transport and catabolism
eggNOG descriptionNAD(P)-binding Rossmann-like domain
Orthologous groupCOG1233
Gene Ontology (8) GO:0005575, GO:0005622, GO:0005623, GO:0005737, GO:0005829, GO:0044424, GO:0044444, GO:0044464

Orthology-based transfer (eggNOG 5.0.2, diamond). EC/KO/GO/CAZy are computed annotations, not manual curation; cross-check against the primary literature before treating a specific reaction as established.

Conservation & selection (intra-MTBC, 145 209 strains)

pN/pS 0.678 · relaxed/neutral
Polymorphic sites (≥ 0.1% of strains) 6 synonymous, 11 missense, 0 nonsense, 2 frameshift
Disruption 2 distinct premature-stop/frameshift site(s); most common in 0.62% of strains (899) · clonal

pN/pS from segregating SNPs (singletons removed) normalised by possible sites. Low pN/pS = purifying selection (a strong signal that a "hypothetical" is a real, constrained gene). A high pN/pS is ambiguous: relaxed constraint or positive selection (drug resistance, antigenic variation) inflate it; e.g. rpoB/katG/pncA score high here for resistance, not loss of function. A clonal disruption (one allele over a clade) suggests lineage pseudogenisation; a convergent one (many independent alleles) is typical of resistance loss-of-function.

Domains (Pfam, hmmscan --cut_ga)

PfamAccessioni-EvalueResiduesDescription
FAD_binding_3PF01494.26 6.6e-068–42 FAD binding domain
Thi4PF01946.24 3.3e-068–52 Thi4 family
FAD_oxidoredPF12831.14 5.8e-079–48 FAD dependent oxidoreductase
GIDAPF01134.29 8.9e-069–54 Glucose inhibited division protein A
FAD_binding_2PF00890.31 4.8e-059–47 FAD binding domain
NAD_binding_8PF13450.13 1.1e-0912–57 NAD(P)-binding Rossmann-like domain

Functional interaction network (STRING v12, guilt-by-association)

Closest characterised functional partner: arfA (peptidoglycan-binding protein ArfA), high confidence from genomic context alone (score 759 excluding text-mining).

PartnerProductScoreNo text-miningChannels (≥400)
Rv0898c hyp hypothetical protein 851 851 ctx neighborhood:847
Rv0899 arfA peptidoglycan-binding protein ArfA 759 759 ctx neighborhood:758
Rv3397c phyA phytoene synthase 759 666 coexpression:654
Rv0901 arfC membrane protein 519 519 ctx neighborhood:514
Rv2850c magnesium chelatase 561 496 coexpression:433
Rv0900 arfB membrane protein 488 488 ctx neighborhood:485
Rv0958 magnesium chelatase 403 367
Rv2062c cobN cobalamin biosynthesis protein CobN 414 359

STRING combines evidence channels (neighborhood, fusion, cooccurrence, coexpression, experimental, database, text-mining) into a 0–1000 score. The ctx badge marks edges carried by the genomic-context channels (conserved neighborhood, fusion, phylogenetic co-occurrence), which are independent of orthology and structure and the strongest signal for an unknown gene. The no text-mining column recomputes the score from data alone, so a link that does not depend on the literature is visible. Association is a function hypothesis, not proof: corroborate with the operon context and the primary literature before assigning a function.

Evidence

  • Legacy H37Rv annotation: oxidoreductase
  • MTBC0 PGAP product: NAD(P)/FAD-dependent oxidoreductase
  • Pfam (hmmscan --cut_ga): FAD_binding_3 PF01494.26 (E=7e-06), Thi4 PF01946.24 (E=3e-06), FAD_oxidored PF12831.14 (E=6e-07), GIDA PF01134.29 (E=9e-06), FAD_binding_2 PF00890.31 (E=5e-05), NAD_binding_8 PF13450.13 (E=1e-09)
  • (auto-curated by rules from PGAP + Pfam + Foldseek; not hand-reviewed)

Sources

  • Ancestral sequence & coordinates: Harrison LB et al. (2024), An imputed ancestral reference genome for the MTBC, doi:10.1101/2023.09.07.556366
  • Product annotation: NCBI PGAP on MTBC0; legacy from H37Rv NC_000962.3 (RefSeq NP_215412.1)
  • Domains: Pfam-A via hmmscan --cut_ga — FAD_binding_3 (PF01494.26), Thi4 (PF01946.24), FAD_oxidored (PF12831.14), GIDA (PF01134.29), FAD_binding_2 (PF00890.31), NAD_binding_8 (PF13450.13)
  • Sequence-level signal: ESM Atlas (EvolutionaryScale × BioHub) — exploratory
  • Controlled vocabulary: eggNOG-mapper 2.1.12 (Cantalapiedra et al. 2021, doi:10.1093/molbev/msab293), eggNOG 5.0 DB (Huerta-Cepas et al. 2019) — OG COG1233
  • Curated reference: UniProt P9WKP7 (SwissProt, reviewed; Evidence at protein level)
  • Intra-MTBC selection: pN/pS and disruption from SPDI variants of 145 209 MTBC strains (this work, local collection vs H37Rv NC_000962.3)
  • Interaction network: STRING v12.0 (Szklarczyk et al. 2023, doi:10.1093/nar/gkac1000), taxon 83332, CC-BY 4.0 — 8 functional partner(s); context anchor arfA
  • Primary literature: none located yet; annotation rests on the domain/homology sources above.

Ancestral MTBC0 protein sequence

>mtbc0_000951|Rv0897c|
MSDHDRDFDVVVVGGGHNGLVAAAYLARAGLRVRLLERLAQTGGAAVSIQAFDGVEVALSRYSYLVSLLPSRIVADLGAPVRLARRPFSSYTPAPATAGRSGLLIGPTGEPRAAHLAAIGAAPDAHGFAAFYRRCRLVTARLWPTLIEPLRTREQARRDIVEYGGHEAAAAWQAMVDEPIGHAIAGAVANDLLRGVIATDALIGTFARMHEPSLMQNICFLYHLVGGGTGVWHVPIGGMGSVTSALATAAARHGAEIVTGADVFALDPDGTVRYHSDGSDGAEHLVRGRFVLVGVTPAVLASLLGEPVAALAPGAQVKVNMVVRRLPRLRDDSVTPQQAFAGTFHVNETWSQLDAAYSQAASGRLPDPLPCEAYCHSLTDPSILSARLRDAGAQTLTVFGLHTPHSVFGDTEGLAERLTAAVLASLNSVLAEPIQDVLWTDAQSKPCIETTTTLDLQRTLGMTGGNIFHGALSWPFADNDDPLDTPARQWGVATDHERIMLCGSGARRGGAVSGIGGHNAAMAVLACLASRRKSP