aglA Family assigned · medium auto-curated

H37Rv Rv2471 · MTBC0 mtbc0_002633 · 546 aa · 2797812–2799452 (+) · RefSeq NP_216987.1

Annotation: from legacy to revised

Legacy (H37Rv / Mycobrowser)alpha-glucosidase AglA
MTBC0 PGAP re-annotationglycoside hydrolase family 13 protein
Revised (this work)Glycoside hydrolase family 13 protein. Pfam: Alpha-amylase (PF00128.32).

Auto-curated: this verdict and function were generated by rules from PGAP + Pfam + Foldseek and have not been hand-reviewed.

Curated reference (UniProt)

UniProt O53198 TrEMBL · unreviewed · Evidence at protein level
UniProt nameProbable alpha-glucosidase AglA

Functional vocabulary (eggNOG-mapper, orthology transfer)

COG category G Carbohydrate transport and metabolism
Preferred nameaglA
eggNOG descriptionalpha amylase, catalytic
Orthologous groupCOG0366
EC number EC 3.2.1.20
KEGG orthology K01187
KEGG pathways map00052, map00500, map01100
CAZy family GH31

Orthology-based transfer (eggNOG 5.0.2, diamond). EC/KO/GO/CAZy are computed annotations, not manual curation; cross-check against the primary literature before treating a specific reaction as established.

Conservation & selection (intra-MTBC, 145 209 strains)

pN/pS 0.454 · purifying
Polymorphic sites (≥ 0.1% of strains) 8 synonymous, 11 missense, 0 nonsense, 0 frameshift

pN/pS from segregating SNPs (singletons removed) normalised by possible sites. Low pN/pS = purifying selection (a strong signal that a "hypothetical" is a real, constrained gene). A high pN/pS is ambiguous: relaxed constraint or positive selection (drug resistance, antigenic variation) inflate it; e.g. rpoB/katG/pncA score high here for resistance, not loss of function. A clonal disruption (one allele over a clade) suggests lineage pseudogenisation; a convergent one (many independent alleles) is typical of resistance loss-of-function.

Domains (Pfam, hmmscan --cut_ga)

PfamAccessioni-EvalueResiduesDescription
Alpha-amylasePF00128.32 9.4e-10739–482 Alpha amylase, catalytic domain

Functional interaction network (STRING v12, guilt-by-association)

Closest characterised functional partner: mak (maltokinase), high confidence from genomic context alone (score 994 excluding text-mining).

PartnerProductScoreNo text-miningChannels (≥400)
Rv0127 mak maltokinase 994 994 ctx fusion:834 experimental:484 database:900
Rv2470 glbO hemoglobin GlbO 950 950 ctx neighborhood:881 fusion:596
Rv0126 treS trehalose synthase/amylase TreS 936 935 database:900
Rv1781c malQ 4-alpha-glucanotransferase 954 922 database:900 textmining:439
Rv0186 bglS beta-glucosidase BglS 945 922 database:900
Rv2469c hyp hypothetical protein 774 774 ctx neighborhood:773
Rv1979c permease 777 766 experimental:451 database:577
Rv2320c rocE cationic amino acid transporter permease RocE 776 765 experimental:451 database:577
Rv3253c cationic amino acid transport integral membrane protein 774 763 experimental:451 database:577
Rv2690c integral membrane protein 774 763 experimental:451 database:577
Rv1999c transporter 774 763 experimental:451 database:577
Rv2468A hyp hypothetical protein 742 742 ctx neighborhood:742
Rv2468c hyp hypothetical protein 742 742 ctx neighborhood:742
Rv2472 hyp hypothetical protein 692 693 ctx neighborhood:691
Rv2006 otsB1 trehalose-6-phosphate phosphatase OtsB 785 635 coexpression:410 textmining:436

STRING combines evidence channels (neighborhood, fusion, cooccurrence, coexpression, experimental, database, text-mining) into a 0–1000 score. The ctx badge marks edges carried by the genomic-context channels (conserved neighborhood, fusion, phylogenetic co-occurrence), which are independent of orthology and structure and the strongest signal for an unknown gene. The no text-mining column recomputes the score from data alone, so a link that does not depend on the literature is visible. Association is a function hypothesis, not proof: corroborate with the operon context and the primary literature before assigning a function.

Evidence

  • Legacy H37Rv annotation: alpha-glucosidase AglA
  • MTBC0 PGAP product: glycoside hydrolase family 13 protein
  • Pfam (hmmscan --cut_ga): Alpha-amylase PF00128.32 (E=9e-107)
  • (auto-curated by rules from PGAP + Pfam + Foldseek; not hand-reviewed)

Sources

  • Ancestral sequence & coordinates: Harrison LB et al. (2024), An imputed ancestral reference genome for the MTBC, doi:10.1101/2023.09.07.556366
  • Product annotation: NCBI PGAP on MTBC0; legacy from H37Rv NC_000962.3 (RefSeq NP_216987.1)
  • Domains: Pfam-A via hmmscan --cut_ga — Alpha-amylase (PF00128.32)
  • Sequence-level signal: ESM Atlas (EvolutionaryScale × BioHub) — exploratory
  • Controlled vocabulary: eggNOG-mapper 2.1.12 (Cantalapiedra et al. 2021, doi:10.1093/molbev/msab293), eggNOG 5.0 DB (Huerta-Cepas et al. 2019) — OG COG0366
  • Curated reference: UniProt O53198 (TrEMBL, unreviewed; Evidence at protein level)
  • Intra-MTBC selection: pN/pS and disruption from SPDI variants of 145 209 MTBC strains (this work, local collection vs H37Rv NC_000962.3)
  • Interaction network: STRING v12.0 (Szklarczyk et al. 2023, doi:10.1093/nar/gkac1000), taxon 83332, CC-BY 4.0 — 35 functional partner(s); context anchor mak
  • Primary literature: none located yet; annotation rests on the domain/homology sources above.

Ancestral MTBC0 protein sequence

>mtbc0_002633|Rv2471|aglA
MDQHQRPDPMGPGSPRASARRPEPDPMGEPWWSRAVFYQVYPRSFADSNGDGVGDLDGLASRLDHLQQLGVDAIWINPVTVSPMADHGYDVADPRDIDPLFGGMPAFERLVAAAHRQGIKVTMDVVPNHTSSAHPWFQAALADLPGSPARDRYFFRDGRGPDGSLPPNNWESVFGGPAWTRVREPDGNPGQWYLHLFDTEQPDLNWDNPEILDDFEKTLRFWLDRGVDGFRIDVAHGMAKPPGLPDSPDLGIEVLHHRDDDPRFNHPNVHAIHRDIRTVIDEYPGAVTVGEVWVHDNARWAEYLRPDELHLGFNFRLARTEFDAAEIRDAVANSLAAAALQNATPTWTLANHDVGREVSRYGGGEIGLRRAKAMAVVMLALPGVVFLYNGQELGLPDVDLPDEVLQDPTWERSGRTERGRDGCRVPIPWSGNIPPFGFSTCPDTWLPMPPEWAALTAEKQRADAGSTLSFFRLALRLRRERNEFDGDVDWLAAPDDALIFRRHGGGLVCALNAAERPLALPAGEPILASAPLTDATLPPNAAAWLV