atsD Resolved · high auto-curated

H37Rv Rv0663 · MTBC0 mtbc0_000701 · 787 aa · 760223–762586 (+) · RefSeq NP_215177.1

Annotation: from legacy to revised

Legacy (H37Rv / Mycobrowser)arylsulfatase AtsD
MTBC0 PGAP re-annotationarylsulfatase AtsD
Revised (this work)Arylsulfatase AtsD. Pfam: Sulfatase (PF00884.29).

Auto-curated: this verdict and function were generated by rules from PGAP + Pfam + Foldseek and have not been hand-reviewed.

Curated reference (UniProt)

UniProt I6XVW9 TrEMBL · unreviewed · Evidence at protein level
UniProt namePossible arylsulfatase AtsD

Functional vocabulary (eggNOG-mapper, orthology transfer)

COG category P Inorganic ion transport and metabolism
Preferred nameatsD
eggNOG descriptionthought to play an important role in the mineralization of sulfates catalytic activity a phenol sulfate H2O a phenol sulfate
Orthologous groupCOG3119
EC number EC 3.1.6.1
KEGG orthology K01130
KEGG pathways map00140, map00600
Gene Ontology (93) GO:0000323, GO:0001775, GO:0002252, GO:0002263, GO:0002274, GO:0002275, GO:0002283, GO:0002366, GO:0002376, GO:0002443, GO:0002444, GO:0002446 +81 more

Orthology-based transfer (eggNOG 5.0.2, diamond). EC/KO/GO/CAZy are computed annotations, not manual curation; cross-check against the primary literature before treating a specific reaction as established.

Conservation & selection (intra-MTBC, 145 209 strains)

pN/pS 0.439 · purifying
Polymorphic sites (≥ 0.1% of strains) 12 synonymous, 17 missense, 0 nonsense, 4 frameshift
Disruption 4 distinct premature-stop/frameshift site(s); most common in 0.84% of strains (1218) · convergent

pN/pS from segregating SNPs (singletons removed) normalised by possible sites. Low pN/pS = purifying selection (a strong signal that a "hypothetical" is a real, constrained gene). A high pN/pS is ambiguous: relaxed constraint or positive selection (drug resistance, antigenic variation) inflate it; e.g. rpoB/katG/pncA score high here for resistance, not loss of function. A clonal disruption (one allele over a clade) suggests lineage pseudogenisation; a convergent one (many independent alleles) is typical of resistance loss-of-function.

Domains (Pfam, hmmscan --cut_ga)

PfamAccessioni-EvalueResiduesDescription
SulfatasePF00884.29 2.7e-6743–459 Sulfatase

Functional interaction network (STRING v12, guilt-by-association)

Closest characterised functional partner: vapC8 (ribonuclease VapC8), high confidence from genomic context alone (score 709 excluding text-mining).

PartnerProductScoreNo text-miningChannels (≥400)
Rv0712 hyp hypothetical protein 841 841 ctx cooccurence:712 experimental:430
Rv0665 vapC8 ribonuclease VapC8 709 709 ctx neighborhood:700
Rv0664 vapB8 antitoxin VapB8 707 708 ctx neighborhood:700
Rv0666 membrane protein 671 672 ctx neighborhood:668
Rv1213 glgC glucose-1-phosphate adenylyltransferase 606 602 coexpression:602
Rv0661c vapC7 ribonuclease VapC7 542 543 ctx neighborhood:540
Rv0662c vapB7 antitoxin VapB7 541 541 ctx neighborhood:540
Rv0342 iniA isoniazid inductible protein IniA 520 520 database:517
Rv0296c sulfatase 517 518 ctx cooccurence:515
Rv2940c mas multifunctional mycocerosic acid synthase 531 501 coexpression:410
Rv2933 ppsC phthiocerol synthesis polyketide synthase type I PpsC 528 499 coexpression:407
Rv1527c pks5 polyketide synthase 527 497 coexpression:405
Rv2048c pks12 polyketide synthase 525 495 coexpression:403
Rv1663 pks17 polyketide synthase 513 495 coexpression:410
Rv3825c pks2 phthioceranic/hydroxyphthioceranic acid synthase 524 494 coexpression:402

STRING combines evidence channels (neighborhood, fusion, cooccurrence, coexpression, experimental, database, text-mining) into a 0–1000 score. The ctx badge marks edges carried by the genomic-context channels (conserved neighborhood, fusion, phylogenetic co-occurrence), which are independent of orthology and structure and the strongest signal for an unknown gene. The no text-mining column recomputes the score from data alone, so a link that does not depend on the literature is visible. Association is a function hypothesis, not proof: corroborate with the operon context and the primary literature before assigning a function.

Evidence

  • Legacy H37Rv annotation: arylsulfatase AtsD
  • MTBC0 PGAP product: arylsulfatase AtsD
  • Pfam (hmmscan --cut_ga): Sulfatase PF00884.29 (E=3e-67)
  • (auto-curated by rules from PGAP + Pfam + Foldseek; not hand-reviewed)

Sources

  • Ancestral sequence & coordinates: Harrison LB et al. (2024), An imputed ancestral reference genome for the MTBC, doi:10.1101/2023.09.07.556366
  • Product annotation: NCBI PGAP on MTBC0; legacy from H37Rv NC_000962.3 (RefSeq NP_215177.1)
  • Domains: Pfam-A via hmmscan --cut_ga — Sulfatase (PF00884.29)
  • Sequence-level signal: ESM Atlas (EvolutionaryScale × BioHub) — exploratory
  • Controlled vocabulary: eggNOG-mapper 2.1.12 (Cantalapiedra et al. 2021, doi:10.1093/molbev/msab293), eggNOG 5.0 DB (Huerta-Cepas et al. 2019) — OG COG3119
  • Curated reference: UniProt I6XVW9 (TrEMBL, unreviewed; Evidence at protein level)
  • Intra-MTBC selection: pN/pS and disruption from SPDI variants of 145 209 MTBC strains (this work, local collection vs H37Rv NC_000962.3)
  • Interaction network: STRING v12.0 (Szklarczyk et al. 2023, doi:10.1093/nar/gkac1000), taxon 83332, CC-BY 4.0 — 72 functional partner(s); context anchor vapC8
  • Primary literature: none located yet; annotation rests on the domain/homology sources above.

Ancestral MTBC0 protein sequence

>mtbc0_000701|Rv0663|atsD
MPQPRTHLPIPSAARTGLITYDAKDPDSTYPPIEQLRPPAGAPNVLLILLDDVGFGASSAFGGPCRTSTAELLAGNGLRYNRFHTTALCSPTRQALLTGRNHHSAGMGGITEIATGAPGYSSVLPNTMSPIARTLKLNGYNTAQFGKCHEVPVWQTSPVGPFDAWPSGGGGFEYFYGFIGGEANQWYPSLYEGTTPVEVNRTPEEGYHFMADMTDKALGWIGQQKALAPDRPFFVYFAPGATHAPHHVPREWADKYRGRFDVGWDALREETFARQKELGVIPADCQLTARHAEIPAWDDMPEDLKPVLCRQMEVYAGFLEYTDHHVGRLVDGLQRLGVLDDTLVFYIIGDNGASAEGTINGTYNEMLNFNGLADIETPRFMTDRLDKFGGPESYNHYSVGWAHAMDTPYQWTKQVASHWGGTRNGTIVHWPNGIAAKGEMRWQFHHVIDVAPTILEAAGLPEPLFVNGVQQHPIEGVSMAYSFDDAQAPDRHETQYFEMFGNRGIYHKGWTAVTKHKTPWILVGEQTVAFDDDVWELYDTTKDWSQAKDLAKEMPEKLHELQRLWLIEATRYNVLPLDDDTASRINPDLAGRPVLIRGNTQVLFSNMGRLSENCVLNLKNKSHTVTAEVEVPETGAEGVIVAQGASIGGWSLYANDGKLKYCYNLGGIKHFYAESADPLPAGAHQVRMEFAYAGGGLGKGGEVTLYVDGQQVGEGHVEATLAIVFSADDGCDVGMDSGSPVSPDYAPGSNAFNGRIKGVQLAIAEAAAAAGHLVDPEHAIRIALARQ