pgk Resolved · high auto-curated

H37Rv Rv1437 · MTBC0 mtbc0_001537 · 412 aa · 1623694–1624932 (+) · RefSeq NP_215953.1

Annotation: from legacy to revised

Legacy (H37Rv / Mycobrowser)phosphoglycerate kinase
MTBC0 PGAP re-annotationphosphoglycerate kinase
Revised (this work)Phosphoglycerate kinase. Pfam: PGK (PF00162.25).

Auto-curated: this verdict and function were generated by rules from PGAP + Pfam + Foldseek and have not been hand-reviewed.

Curated reference (UniProt)

UniProt P9WID1 SwissProt · reviewed · Evidence at protein level
UniProt namePhosphoglycerate kinase
EC (curated) EC 2.7.2.3

Functional vocabulary (eggNOG-mapper, orthology transfer)

COG category F Nucleotide transport and metabolism
Preferred namepgk
eggNOG descriptionBelongs to the phosphoglycerate kinase family
Orthologous groupCOG0126
EC number EC 2.7.2.3, EC 5.3.1.1
KEGG orthology K00927, K01803
KEGG pathways map00010, map00051, map00562, map00710, map01100, map01110, map01120, map01130, map01200, map01230
KEGG modules M00001, M00002, M00003, M00165, M00166, M00308, M00552
Gene Ontology (12) GO:0005575, GO:0005576, GO:0005622, GO:0005623, GO:0005737, GO:0005886, GO:0008150, GO:0016020, GO:0040007, GO:0044424, GO:0044464, GO:0071944

Orthology-based transfer (eggNOG 5.0.2, diamond). EC/KO/GO/CAZy are computed annotations, not manual curation; cross-check against the primary literature before treating a specific reaction as established.

Conservation & selection (intra-MTBC, 145 209 strains)

pN/pS 0.727 · relaxed/neutral
Polymorphic sites (≥ 0.1% of strains) 1 synonymous, 2 missense, 0 nonsense, 0 frameshift

pN/pS from segregating SNPs (singletons removed) normalised by possible sites. Low pN/pS = purifying selection (a strong signal that a "hypothetical" is a real, constrained gene). A high pN/pS is ambiguous: relaxed constraint or positive selection (drug resistance, antigenic variation) inflate it; e.g. rpoB/katG/pncA score high here for resistance, not loss of function. A clonal disruption (one allele over a clade) suggests lineage pseudogenisation; a convergent one (many independent alleles) is typical of resistance loss-of-function.

Domains (Pfam, hmmscan --cut_ga)

PfamAccessioni-EvalueResiduesDescription
PGKPF00162.25 1.0e-13414–394 Phosphoglycerate kinase

Functional interaction network (STRING v12, guilt-by-association)

Closest characterised functional partner: tpi (triosephosphate isomerase), high confidence from genomic context alone (score 1000 excluding text-mining).

PartnerProductScoreNo text-miningChannels (≥400)
Rv1438 tpi triosephosphate isomerase 999 1000 ctx neighborhood:882 fusion:900 cooccurence:656 coexpression:963 textmining:832
Rv1436 gap glyceraldehyde 3-phosphate dehydrogenase 999 1000 ctx neighborhood:882 cooccurence:731 coexpression:920 experimental:464 database:900 textmining:807
Rv1023 eno enolase 995 985 ctx cooccurence:685 coexpression:858 experimental:665 textmining:733
Rv0489 gpm1 2,3-bisphosphoglycerate-dependent phosphoglycerate mutase 990 975 coexpression:727 database:900 textmining:648
Rv1617 pykA pyruvate kinase 962 902 ctx cooccurence:561 coexpression:738 textmining:630
Rv0946c pgi glucose-6-phosphate isomerase 975 888 coexpression:853 textmining:793
Rv0363c fba fructose-bisphosphate aldolase 958 884 coexpression:827 textmining:653
Rv1408 rpe ribulose-phosphate 3-epimerase 925 798 ctx cooccurence:676 textmining:649
Rv1449c tkt transketolase 913 762 coexpression:653 textmining:652
Rv3010c pfkA 6-phosphofructokinase 880 748 coexpression:652 textmining:545
Rv1392 metK S-adenosylmethionine synthetase 787 722 ctx cooccurence:566
Rv2614c thrS threonine--tRNA ligase 737 712 ctx cooccurence:625
Rv1732c hyp hypothetical protein 661 661 ctx fusion:641
Rv1389 gmk guanylate kinase 675 623 ctx cooccurence:574
Rv0684 fusA1 elongation factor G 710 622 coexpression:434

STRING combines evidence channels (neighborhood, fusion, cooccurrence, coexpression, experimental, database, text-mining) into a 0–1000 score. The ctx badge marks edges carried by the genomic-context channels (conserved neighborhood, fusion, phylogenetic co-occurrence), which are independent of orthology and structure and the strongest signal for an unknown gene. The no text-mining column recomputes the score from data alone, so a link that does not depend on the literature is visible. Association is a function hypothesis, not proof: corroborate with the operon context and the primary literature before assigning a function.

Evidence

  • Legacy H37Rv annotation: phosphoglycerate kinase
  • MTBC0 PGAP product: phosphoglycerate kinase
  • Pfam (hmmscan --cut_ga): PGK PF00162.25 (E=1e-134)
  • (auto-curated by rules from PGAP + Pfam + Foldseek; not hand-reviewed)

Sources

  • Ancestral sequence & coordinates: Harrison LB et al. (2024), An imputed ancestral reference genome for the MTBC, doi:10.1101/2023.09.07.556366
  • Product annotation: NCBI PGAP on MTBC0; legacy from H37Rv NC_000962.3 (RefSeq NP_215953.1)
  • Domains: Pfam-A via hmmscan --cut_ga — PGK (PF00162.25)
  • Sequence-level signal: ESM Atlas (EvolutionaryScale × BioHub) — exploratory
  • Controlled vocabulary: eggNOG-mapper 2.1.12 (Cantalapiedra et al. 2021, doi:10.1093/molbev/msab293), eggNOG 5.0 DB (Huerta-Cepas et al. 2019) — OG COG0126
  • Curated reference: UniProt P9WID1 (SwissProt, reviewed; Evidence at protein level)
  • Intra-MTBC selection: pN/pS and disruption from SPDI variants of 145 209 MTBC strains (this work, local collection vs H37Rv NC_000962.3)
  • Interaction network: STRING v12.0 (Szklarczyk et al. 2023, doi:10.1093/nar/gkac1000), taxon 83332, CC-BY 4.0 — 158 functional partner(s); context anchor tpi
  • Primary literature: none located yet; annotation rests on the domain/homology sources above.

Ancestral MTBC0 protein sequence

>mtbc0_001537|Rv1437|pgk
MSVANLKDLLAEGVSGRGVLVRSDLNVPLDEDGTITDAGRIIASAPTLKALLDADAKVVVAAHLGRPKDGPDPTLSLAPVAVALGEQLGRHVQLAGDVVGADALARAEGLTGGDILLLENIRFDKRETSKNDDDRRALAKQLVELVGTGGVFVSDGFGVVHRKQASVYDIATLLPHYAGTLVADEMRVLEQLTSSTQRPYAVVLGGSKVSDKLGVIESLATKADSIVIGGGMCFTFLAAQGFSVGTSLLEDDMIEVCRGLLETYHDVLRLPVDLVVTEKFAADSPPQTVDVGAVPNGLMGLDIGPGSIKRFSTLLSNAGTIFWNGPMGVFEFPAYAAGTRGVAEAIVAATGKGAFSVVGGGDSAAAVRAMNIPEGAFSHISTGGGASLEYLEGKTLPGIEVLSREQPTGGVL