lpqW Resolved · high auto-curated

H37Rv Rv1166 · MTBC0 mtbc0_001255 · 635 aa · 1304593–1306500 (+) · RefSeq NP_215682.1

Annotation: from legacy to revised

Legacy (H37Rv / Mycobrowser)monoacyl phosphatidylinositol tetramannoside-binding protein LpqW
MTBC0 PGAP re-annotationlipoarabinomannan biosynthesis protein LpqW
Revised (this work)Lipoarabinomannan biosynthesis protein LpqW. Pfam: SBP_bac_5 (PF00496.28).

Auto-curated: this verdict and function were generated by rules from PGAP + Pfam + Foldseek and have not been hand-reviewed.

Curated reference (UniProt)

UniProt P9WGU7 SwissProt · reviewed · Evidence at protein level
UniProt nameProbable monoacyl phosphatidylinositol tetramannoside-binding protein LpqW
Curated functionMay directly or indirectly regulate the accessibility of the key branch point intermediate, monoacyl phosphatidylinositol tetramannoside (AcPIM4), to the elongating alpha-1,6 mannosyltransferases which could regulate the lipoarabinomannans (LAMs) biosynthesis.

Functional vocabulary (eggNOG-mapper, orthology transfer)

COG category E Amino acid transport and metabolism
Preferred namelpqW
eggNOG descriptionMonoacyl phosphatidylinositol tetramannoside-binding protein
Orthologous groupCOG0747
Gene Ontology (28) GO:0005575, GO:0005576, GO:0005623, GO:0006629, GO:0006643, GO:0006664, GO:0008150, GO:0008152, GO:0008610, GO:0009058, GO:0009247, GO:0009987 +16 more

Orthology-based transfer (eggNOG 5.0.2, diamond). EC/KO/GO/CAZy are computed annotations, not manual curation; cross-check against the primary literature before treating a specific reaction as established.

Conservation & selection (intra-MTBC, 145 209 strains)

pN/pS 0.362 · purifying
Polymorphic sites (≥ 0.1% of strains) 4 synonymous, 4 missense, 0 nonsense, 0 frameshift

pN/pS from segregating SNPs (singletons removed) normalised by possible sites. Low pN/pS = purifying selection (a strong signal that a "hypothetical" is a real, constrained gene). A high pN/pS is ambiguous: relaxed constraint or positive selection (drug resistance, antigenic variation) inflate it; e.g. rpoB/katG/pncA score high here for resistance, not loss of function. A clonal disruption (one allele over a clade) suggests lineage pseudogenisation; a convergent one (many independent alleles) is typical of resistance loss-of-function.

Domains (Pfam, hmmscan --cut_ga)

PfamAccessioni-EvalueResiduesDescription
SBP_bac_5PF00496.28 2.6e-31119–485 Bacterial extracellular solute-binding proteins, family 5 Middle

Functional interaction network (STRING v12, guilt-by-association)

Closest characterised functional partner: typA (GTP-binding translation elongation factor), high confidence from genomic context alone (score 901 excluding text-mining).

PartnerProductScoreNo text-miningChannels (≥400)
Rv1165 typA GTP-binding translation elongation factor 900 901 ctx neighborhood:773 coexpression:580
Rv1632c hyp hypothetical protein 778 778 ctx cooccurence:721
Rv3850 hyp hypothetical protein 769 769 ctx cooccurence:767
Rv0556 transmembrane protein 768 768 ctx cooccurence:767
Rv3438 hyp hypothetical protein 767 768 ctx cooccurence:767
Rv3212 hyp hypothetical protein 775 767 ctx cooccurence:764
Rv2732c transmembrane protein 767 767 ctx cooccurence:765
Rv1164 narI nitrate reductase subunit gamma 767 767 ctx neighborhood:755
Rv1163 narJ nitrate reductase subunit delta 766 766 ctx neighborhood:755
Rv1109c hyp hypothetical protein 765 766 ctx cooccurence:765
Rv0383c ttfA hyp hypothetical protein 763 763 ctx cooccurence:759
Rv2446c integral membrane protein 756 757 ctx cooccurence:755
Rv0817c lmeA hyp hypothetical protein 751 751 ctx cooccurence:751
Rv0358 hyp hypothetical protein 748 748 ctx cooccurence:744
Rv0996 transmembrane protein 748 748 ctx cooccurence:747

STRING combines evidence channels (neighborhood, fusion, cooccurrence, coexpression, experimental, database, text-mining) into a 0–1000 score. The ctx badge marks edges carried by the genomic-context channels (conserved neighborhood, fusion, phylogenetic co-occurrence), which are independent of orthology and structure and the strongest signal for an unknown gene. The no text-mining column recomputes the score from data alone, so a link that does not depend on the literature is visible. Association is a function hypothesis, not proof: corroborate with the operon context and the primary literature before assigning a function.

Evidence

  • Legacy H37Rv annotation: monoacyl phosphatidylinositol tetramannoside-binding protein LpqW
  • MTBC0 PGAP product: lipoarabinomannan biosynthesis protein LpqW
  • Pfam (hmmscan --cut_ga): SBP_bac_5 PF00496.28 (E=3e-31)
  • (auto-curated by rules from PGAP + Pfam + Foldseek; not hand-reviewed)

Sources

  • Ancestral sequence & coordinates: Harrison LB et al. (2024), An imputed ancestral reference genome for the MTBC, doi:10.1101/2023.09.07.556366
  • Product annotation: NCBI PGAP on MTBC0; legacy from H37Rv NC_000962.3 (RefSeq NP_215682.1)
  • Domains: Pfam-A via hmmscan --cut_ga — SBP_bac_5 (PF00496.28)
  • Sequence-level signal: ESM Atlas (EvolutionaryScale × BioHub) — exploratory
  • Controlled vocabulary: eggNOG-mapper 2.1.12 (Cantalapiedra et al. 2021, doi:10.1093/molbev/msab293), eggNOG 5.0 DB (Huerta-Cepas et al. 2019) — OG COG0747
  • Curated reference: UniProt P9WGU7 (SwissProt, reviewed; Evidence at protein level)
  • Intra-MTBC selection: pN/pS and disruption from SPDI variants of 145 209 MTBC strains (this work, local collection vs H37Rv NC_000962.3)
  • Interaction network: STRING v12.0 (Szklarczyk et al. 2023, doi:10.1093/nar/gkac1000), taxon 83332, CC-BY 4.0 — 136 functional partner(s); context anchor typA
  • Primary literature: none located yet; annotation rests on the domain/homology sources above.

Ancestral MTBC0 protein sequence

>mtbc0_001255|Rv1166|lpqW
MGVPSPVRRVCVTVGALVALACMVLAGCTVSPPPAPQSTDTPRSTPPPPRRPTQIIMGIDWIGPGFNPHLLSDLSPVNAAISALVLPSAFRPIPDPNTPTGSRWEMDPTLLVSADVTNNHPFTVTYKIRPEAQWTDNAPIAADDFWYLWQQMVTQPGVVDPAGYHLITSVQSLEGGKQAVVTFAQPYPAWRELFTDILPAHIVKDIPGGFASGLARALPVTGGQFRVENIDPQRDEILIARNDRYWGPPSKPGIILFRRAGAPAALADSVRNGDTQVAQVHGGSAAFAQLSAIPDVRTARIVTPRVMQFTLRANVPKLADTQVRKAILGLLDVDLLAAVGAGTDNTVTLDQAQIRSPSDPGYVPTAPPAMSSAAALGLLEASGFQVDTNTSVSPAPSVPDSTTTSVSTGPPEVIRGRISKDGEQLTLVIGVAANDPTSVAVANTAADQLRDVGIAATVLALDPVTLYHDALNDNRVDAIVGWRQAGGNLATLLASRYGCPALQATTVPAANAPTTAPSAPIGPTPSAAPDTATPPPTAPRRPSDPGALVKAPSNLTGICDRSIQSNIDAALNGTKNINDVITAVEPRLWNMSTVLPILQDTTIVAAGPSVQNVSLSGAVPVGIVGDAGQWVKTGQ