Rv3802c Resolved · high auto-curated

H37Rv Rv3802c · MTBC0 mtbc0_004030 · 336 aa · 4287465–4288475 (-) · RefSeq NP_218319.1

Annotation: from legacy to revised

Legacy (H37Rv / Mycobrowser)membrane protein
MTBC0 PGAP re-annotationphospholipase
Revised (this work)Phospholipase. Pfam: Cutinase (PF01083.29), PE-PPE (PF08237.18).

Auto-curated: this verdict and function were generated by rules from PGAP + Pfam + Foldseek and have not been hand-reviewed.

Curated reference (UniProt)

UniProt O53581 SwissProt · reviewed · Evidence at protein level
UniProt nameCarboxylesterase/lipase Culp6
EC (curated) EC 3.1.1.-
Curated functionShows esterase and phospholipase A activities. May be involved in cell wall biosynthesis and/or maintenance. Can hydrolyze various substrates, including the p-nitrophenol-linked aliphatic esters pNP-laurate (C12), pNP-myristate (C14), pNP-palmitate (C16), pNP-stearate (C18), pNP-butyrate (C4), phosphatidylcholine, phosphatidylethanolamine, phosphatidylserine, 4-methylumbelliferyl heptanoate and palmitic acid and arachidonic acid containing phospholipids. Does not exhibit cutinase activity.

Functional vocabulary (eggNOG-mapper, orthology transfer)

COG category S Function unknown
Preferred nameelrF
eggNOG descriptioncutinase
Orthologous groupCOG4223

Orthology-based transfer (eggNOG 5.0.2, diamond). EC/KO/GO/CAZy are computed annotations, not manual curation; cross-check against the primary literature before treating a specific reaction as established.

Conservation & selection (intra-MTBC, 145 209 strains)

pN/pS 0.848 · relaxed/neutral
Polymorphic sites (≥ 0.1% of strains) 2 synonymous, 5 missense, 0 nonsense, 1 frameshift
Disruption 1 distinct premature-stop/frameshift site(s); most common in 0.26% of strains (384) · clonal

pN/pS from segregating SNPs (singletons removed) normalised by possible sites. Low pN/pS = purifying selection (a strong signal that a "hypothetical" is a real, constrained gene). A high pN/pS is ambiguous: relaxed constraint or positive selection (drug resistance, antigenic variation) inflate it; e.g. rpoB/katG/pncA score high here for resistance, not loss of function. A clonal disruption (one allele over a clade) suggests lineage pseudogenisation; a convergent one (many independent alleles) is typical of resistance loss-of-function.

Domains (Pfam, hmmscan --cut_ga)

PfamAccessioni-EvalueResiduesDescription
CutinasePF01083.29 5.3e-15110–275 Cutinase
PE-PPEPF08237.18 2.7e-07120–310 PE-PPE domain

Functional interaction network (STRING v12, guilt-by-association)

Closest characterised functional partner: fbpD (MPT51/MPB51 antigen), high confidence from genomic context alone (score 888 excluding text-mining).

PartnerProductScoreNo text-miningChannels (≥400)
Rv3803c fbpD MPT51/MPB51 antigen 891 888 ctx neighborhood:737 cooccurence:589
Rv3800c pks13 polyketide synthase 905 874 ctx neighborhood:719 cooccurence:564
Rv3805c aftB terminal beta-(1->2)-arabinofuranosyltransferase 852 848 ctx cooccurence:771
Rv3804c fbpA diacylglycerol acyltransferase/mycolyltransferase Ag85A 833 808 ctx cooccurence:702
Rv3794 embA arabinosyltransferase A 807 808 ctx cooccurence:773
Rv3795 embB arabinosyltransferase B 805 806 ctx cooccurence:771
Rv3793 embC arabinosyltransferase C 805 805 ctx cooccurence:771
Rv3801c fadD32 long-chain-fatty-acid--AMP ligase FadD32 838 779 ctx neighborhood:719
Rv3668c protease 778 779 ctx cooccurence:769
Rv0129c fbpC diacylglycerol acyltransferase/mycolyltransferase Ag85C 800 778 ctx cooccurence:743
Rv3244c lpqB lipoprotein LpqB 774 774 ctx cooccurence:764
Rv2673 aftC alpha-(1->3)-arabinofuranosyltransferase 774 774 ctx cooccurence:774
Rv1476 membrane protein 774 774 ctx cooccurence:773
Rv3035 hyp hypothetical protein 770 771 ctx cooccurence:767
Rv2342 hyp hypothetical protein 761 762 ctx cooccurence:756

STRING combines evidence channels (neighborhood, fusion, cooccurrence, coexpression, experimental, database, text-mining) into a 0–1000 score. The ctx badge marks edges carried by the genomic-context channels (conserved neighborhood, fusion, phylogenetic co-occurrence), which are independent of orthology and structure and the strongest signal for an unknown gene. The no text-mining column recomputes the score from data alone, so a link that does not depend on the literature is visible. Association is a function hypothesis, not proof: corroborate with the operon context and the primary literature before assigning a function.

Evidence

  • Legacy H37Rv annotation: membrane protein
  • MTBC0 PGAP product: phospholipase
  • Pfam (hmmscan --cut_ga): Cutinase PF01083.29 (E=5e-15), PE-PPE PF08237.18 (E=3e-07)
  • (auto-curated by rules from PGAP + Pfam + Foldseek; not hand-reviewed)

Sources

  • Ancestral sequence & coordinates: Harrison LB et al. (2024), An imputed ancestral reference genome for the MTBC, doi:10.1101/2023.09.07.556366
  • Product annotation: NCBI PGAP on MTBC0; legacy from H37Rv NC_000962.3 (RefSeq NP_218319.1)
  • Domains: Pfam-A via hmmscan --cut_ga — Cutinase (PF01083.29), PE-PPE (PF08237.18)
  • Sequence-level signal: ESM Atlas (EvolutionaryScale × BioHub) — exploratory
  • Controlled vocabulary: eggNOG-mapper 2.1.12 (Cantalapiedra et al. 2021, doi:10.1093/molbev/msab293), eggNOG 5.0 DB (Huerta-Cepas et al. 2019) — OG COG4223
  • Curated reference: UniProt O53581 (SwissProt, reviewed; Evidence at protein level)
  • Intra-MTBC selection: pN/pS and disruption from SPDI variants of 145 209 MTBC strains (this work, local collection vs H37Rv NC_000962.3)
  • Interaction network: STRING v12.0 (Szklarczyk et al. 2023, doi:10.1093/nar/gkac1000), taxon 83332, CC-BY 4.0 — 128 functional partner(s); context anchor fbpD
  • Primary literature: none located yet; annotation rests on the domain/homology sources above.

Ancestral MTBC0 protein sequence

>mtbc0_004030|Rv3802c|
MAKNSRRKRHRILAWIAAGAMASVVALVIVAVVIMLRGAESPPSAVPPGVLPPGPTPAHPHKPRPAFQDASCPDVQMISVPGTWESSPQQNPLNPVQFPKALLLKVTGPIAQQFAPARVQTYTVAYTAQFHNPLTTDNQMSYNDSRAEGTRAMVAAMTDMNNRCPLTSYVLIGFSQGAVIAGDVASDIGNGRGPVDEDLVLGVTLIADGRRQQGVGNQVPPSPRGEGAEITLHEVPVLSGLGLTMTGPRPGGFGALDGRTNEICAQGDLICAAPAQAFSPANLPTTLNTLAGGAGQPVHAMYATPEFWNSDGEPATEWTLNWAHQLIENAPHPKHR