Rv2974c Family assigned · medium auto-curated

H37Rv Rv2974c · MTBC0 - · 470 aa · 3329949–3331361 (-) · RefSeq NP_217490.1

Annotation: from legacy to revised

Legacy (H37Rv / Mycobrowser)hypothetical protein
MTBC0 PGAP re-annotation
Revised (this work)Contains Dak2 (PF02734.23), FakA-like_M (PF21645.4), FakA-like_C (PF13684.12) domain(s); putative function inferred from the domain architecture.

Auto-curated: this verdict and function were generated by rules from PGAP + Pfam + Foldseek and have not been hand-reviewed.

Annotated on the H37Rv protein: this gene has no 1:1 ancestral MTBC0 anchor (PE/PPE, paralogue, IS element, or otherwise unanchored CDS).

Curated reference (UniProt)

UniProt I6Y259 TrEMBL · unreviewed · Evidence at protein level
UniProt nameConserved hypothetical alanine rich protein

Functional vocabulary (eggNOG-mapper, orthology transfer)

COG category S Function unknown
Preferred namedak3
eggNOG descriptionDihydroxyacetone kinase
Orthologous groupCOG1461
KEGG orthology K07030

Orthology-based transfer (eggNOG 5.0.2, diamond). EC/KO/GO/CAZy are computed annotations, not manual curation; cross-check against the primary literature before treating a specific reaction as established.

Conservation & selection (intra-MTBC, 145 209 strains)

pN/pS 1.363 · diversifying/relaxed
Polymorphic sites (≥ 0.1% of strains) 2 synonymous, 7 missense, 0 nonsense, 0 frameshift

pN/pS from segregating SNPs (singletons removed) normalised by possible sites. Low pN/pS = purifying selection (a strong signal that a "hypothetical" is a real, constrained gene). A high pN/pS is ambiguous: relaxed constraint or positive selection (drug resistance, antigenic variation) inflate it; e.g. rpoB/katG/pncA score high here for resistance, not loss of function. A clonal disruption (one allele over a clade) suggests lineage pseudogenisation; a convergent one (many independent alleles) is typical of resistance loss-of-function.

Domains (Pfam, hmmscan --cut_ga)

PfamAccessioni-EvalueResiduesDescription
Dak2PF02734.23 7.5e-171–131 DAK2 domain
FakA-like_MPF21645.4 8.0e-15162–232 Fatty acid kinase subunit A-like, middle domain
FakA-like_CPF13684.12 1.9e-49249–470 Fatty acid kinase subunit A-like, C-terminal

Functional interaction network (STRING v12, guilt-by-association)

Closest characterised functional partner: recG (ATP-dependent DNA helicase RecG), high confidence from genomic context alone (score 944 excluding text-mining). This association is the citable seed of a function hypothesis for this hypothetical protein.

PartnerProductScoreNo text-miningChannels (≥400)
Rv2975c hyp hypothetical protein 978 978 ctx neighborhood:773 fusion:875
Rv2973c recG ATP-dependent DNA helicase RecG 944 944 ctx neighborhood:800 coexpression:732
Rv2417c DegV domain-containing protein 930 928 ctx cooccurence:774 experimental:652
Rv2972c hyp hypothetical protein 809 809 ctx neighborhood:800
Rv2185c TB16.3 hyp hypothetical protein 521 521 ctx cooccurence:521
Rv0854 hyp hypothetical protein 515 515 ctx cooccurence:515
Rv0857 hyp hypothetical protein 499 499 ctx cooccurence:496
Rv1696 recN DNA repair protein RecN 440 440 coexpression:440
Rv0474 HTH-type transcriptional regulator 428 428 ctx cooccurence:423
Rv1018c glmU bifunctional UDP-N-acetylglucosamine pyrophosphorylase/glucosamine-1-phosphate N-acetyltransferase 410 410 coexpression:410
Rv0024 NLP/P60 family protein 419 188
Rv2065 cobH precorrin-8X methylmutase 667 79 textmining:654
Rv2070c cobK precorrin-6A reductase 529 78 textmining:511
Rv0105c rpmB1 50S ribosomal protein L28 526 73 textmining:510
Rv2277c glycerolphosphodiesterase 658 47 textmining:656

STRING combines evidence channels (neighborhood, fusion, cooccurrence, coexpression, experimental, database, text-mining) into a 0–1000 score. The ctx badge marks edges carried by the genomic-context channels (conserved neighborhood, fusion, phylogenetic co-occurrence), which are independent of orthology and structure and the strongest signal for an unknown gene. The no text-mining column recomputes the score from data alone, so a link that does not depend on the literature is visible. Association is a function hypothesis, not proof: corroborate with the operon context and the primary literature before assigning a function.

Evidence

  • Annotation from H37Rv (no MTBC0 1:1 anchor; H37Rv protein used): hypothetical protein
  • Pfam (hmmscan --cut_ga): Dak2 PF02734.23 (E=7e-17), FakA-like_M PF21645.4 (E=8e-15), FakA-like_C PF13684.12 (E=2e-49)
  • (auto-curated by rules from PGAP + Pfam + Foldseek; not hand-reviewed)

Sources

  • Ancestral sequence & coordinates: Harrison LB et al. (2024), An imputed ancestral reference genome for the MTBC, doi:10.1101/2023.09.07.556366
  • Product annotation: NCBI PGAP on MTBC0; legacy from H37Rv NC_000962.3 (RefSeq NP_217490.1)
  • Domains: Pfam-A via hmmscan --cut_ga — Dak2 (PF02734.23), FakA-like_M (PF21645.4), FakA-like_C (PF13684.12)
  • Sequence-level signal: ESM Atlas (EvolutionaryScale × BioHub) — exploratory
  • Controlled vocabulary: eggNOG-mapper 2.1.12 (Cantalapiedra et al. 2021, doi:10.1093/molbev/msab293), eggNOG 5.0 DB (Huerta-Cepas et al. 2019) — OG COG1461
  • Curated reference: UniProt I6Y259 (TrEMBL, unreviewed; Evidence at protein level)
  • Intra-MTBC selection: pN/pS and disruption from SPDI variants of 145 209 MTBC strains (this work, local collection vs H37Rv NC_000962.3)
  • Interaction network: STRING v12.0 (Szklarczyk et al. 2023, doi:10.1093/nar/gkac1000), taxon 83332, CC-BY 4.0 — 26 functional partner(s); context anchor recG
  • Primary literature: none located yet; annotation rests on the domain/homology sources above.

Ancestral MTBC0 protein sequence

>H37Rv|Rv2974c|
MNGARGNSGVILSQILRGIAEVTATAAAASGAVLRAVDANALGAALWRGVELVVASMGGVEVPGTIVSVLRAAAGAVDQCAHEGLAGAVTAAGDAAVIALEKTPEQLDVLADAGAVDAGGRGLLVLLDALRSTICGQAPARAVYEPSPRALPTDTATQRPAPQFEVMYLLAVCDAAAADQLRDRLKELGESVAIAAAPPDSYSVHVHTDDAGAAVEAGLAVGRVSRIVISALGSGTSGLPAGGWTRGRAVLAVVDGDGAAELFAGEGACVLRPGPDAVTPAADISAHQLVRAVVDTGAAHVMVLPNGYVAAEELVAGCTAAIGWGVDVVPVPTGSMVQGLAALAVHDAARQAVDDGYSMARAAGASRHGSVRIATQKALTWAGTCKPGDGLGIAGDEVLIVADDVAAAAIGLVDLLLASGGDLVTVLIGAGVTEDVAVVLERHVHDHHPGTELVSYRTGHRGDALLIGVE