proB Resolved · high auto-curated

H37Rv Rv2439c · MTBC0 mtbc0_002598 · 376 aa · 2761365–2762495 (-) · RefSeq NP_216955.1

Annotation: from legacy to revised

Legacy (H37Rv / Mycobrowser)glutamate 5-kinase protein
MTBC0 PGAP re-annotationglutamate 5-kinase
Revised (this work)Glutamate 5-kinase. Pfam: AA_kinase (PF00696.34), PUA (PF01472.27).

Auto-curated: this verdict and function were generated by rules from PGAP + Pfam + Foldseek and have not been hand-reviewed.

Curated reference (UniProt)

UniProt P9WHU9 SwissProt · reviewed · Evidence at protein level
UniProt nameGlutamate 5-kinase
EC (curated) EC 2.7.2.11
Curated functionCatalyzes the transfer of a phosphate group to glutamate to form L-glutamate 5-phosphate.

Functional vocabulary (eggNOG-mapper, orthology transfer)

COG category E Amino acid transport and metabolism
Preferred nameproB
eggNOG descriptionCatalyzes the transfer of a phosphate group to glutamate to form L-glutamate 5-phosphate
Orthologous groupCOG0263
EC number EC 2.7.2.11
KEGG orthology K00931
KEGG pathways map00330, map00332, map01100, map01130, map01230
KEGG modules M00015
Gene Ontology (51) GO:0003674, GO:0003824, GO:0004349, GO:0005575, GO:0005622, GO:0005623, GO:0005737, GO:0005829, GO:0006082, GO:0006520, GO:0006560, GO:0006561 +39 more

Orthology-based transfer (eggNOG 5.0.2, diamond). EC/KO/GO/CAZy are computed annotations, not manual curation; cross-check against the primary literature before treating a specific reaction as established.

Conservation & selection (intra-MTBC, 145 209 strains)

pN/pS 0.227 · purifying
Polymorphic sites (≥ 0.1% of strains) 7 synonymous, 4 missense, 0 nonsense, 0 frameshift

pN/pS from segregating SNPs (singletons removed) normalised by possible sites. Low pN/pS = purifying selection (a strong signal that a "hypothetical" is a real, constrained gene). A high pN/pS is ambiguous: relaxed constraint or positive selection (drug resistance, antigenic variation) inflate it; e.g. rpoB/katG/pncA score high here for resistance, not loss of function. A clonal disruption (one allele over a clade) suggests lineage pseudogenisation; a convergent one (many independent alleles) is typical of resistance loss-of-function.

Domains (Pfam, hmmscan --cut_ga)

PfamAccessioni-EvalueResiduesDescription
AA_kinasePF00696.34 1.2e-3815–239 Amino acid kinase family
PUAPF01472.27 6.1e-16281–350 PUA domain

Functional interaction network (STRING v12, guilt-by-association)

Closest characterised functional partner: proA (gamma-glutamyl phosphate reductase), high confidence from genomic context alone (score 1000 excluding text-mining).

PartnerProductScoreNo text-miningChannels (≥400)
Rv2427c proA gamma-glutamyl phosphate reductase 999 1000 ctx fusion:900 cooccurence:774 coexpression:806 database:900 textmining:705
Rv2322c rocD1 Rv2322c, (MTCY3G12.12), len: 221 aa. Probable rocD1,ornithine aminotransferase, highly similar to N-terminal region of other ornithine amino 923 915 database:900
Rv2440c obg GTPase Obg 988 904 ctx neighborhood:881 textmining:889
Rv1187 rocA pyrroline-5-carboxylate dehydrogenase RocA 914 902 database:900
Rv0500 proC pyrroline-5-carboxylate reductase 945 832 ctx cooccurence:660 coexpression:422 textmining:688
Rv2441c rpmA 50S ribosomal protein L27 974 783 ctx neighborhood:782 textmining:887
Rv2442c rplU 50S ribosomal protein L21 828 776 ctx neighborhood:775
Rv2444c rne ribonuclease E 562 563 ctx neighborhood:544
Rv3709c ask aspartokinase 548 420
Rv1017c prsA ribose-phosphate pyrophosphokinase 425 388
Rv2192c trpD anthranilate phosphoribosyltransferase 525 379
Rv1294 thrA homoserine dehydrogenase 487 331
Rv0884c serC phosphoserine aminotransferase 482 325
Rv1654 argB acetylglutamate kinase 729 311 textmining:624
Rv2995c leuB 3-isopropylmalate dehydrogenase 418 240

STRING combines evidence channels (neighborhood, fusion, cooccurrence, coexpression, experimental, database, text-mining) into a 0–1000 score. The ctx badge marks edges carried by the genomic-context channels (conserved neighborhood, fusion, phylogenetic co-occurrence), which are independent of orthology and structure and the strongest signal for an unknown gene. The no text-mining column recomputes the score from data alone, so a link that does not depend on the literature is visible. Association is a function hypothesis, not proof: corroborate with the operon context and the primary literature before assigning a function.

Evidence

  • Legacy H37Rv annotation: glutamate 5-kinase protein
  • MTBC0 PGAP product: glutamate 5-kinase
  • Pfam (hmmscan --cut_ga): AA_kinase PF00696.34 (E=1e-38), PUA PF01472.27 (E=6e-16)
  • (auto-curated by rules from PGAP + Pfam + Foldseek; not hand-reviewed)

Sources

  • Ancestral sequence & coordinates: Harrison LB et al. (2024), An imputed ancestral reference genome for the MTBC, doi:10.1101/2023.09.07.556366
  • Product annotation: NCBI PGAP on MTBC0; legacy from H37Rv NC_000962.3 (RefSeq NP_216955.1)
  • Domains: Pfam-A via hmmscan --cut_ga — AA_kinase (PF00696.34), PUA (PF01472.27)
  • Sequence-level signal: ESM Atlas (EvolutionaryScale × BioHub) — exploratory
  • Controlled vocabulary: eggNOG-mapper 2.1.12 (Cantalapiedra et al. 2021, doi:10.1093/molbev/msab293), eggNOG 5.0 DB (Huerta-Cepas et al. 2019) — OG COG0263
  • Curated reference: UniProt P9WHU9 (SwissProt, reviewed; Evidence at protein level)
  • Intra-MTBC selection: pN/pS and disruption from SPDI variants of 145 209 MTBC strains (this work, local collection vs H37Rv NC_000962.3)
  • Interaction network: STRING v12.0 (Szklarczyk et al. 2023, doi:10.1093/nar/gkac1000), taxon 83332, CC-BY 4.0 — 28 functional partner(s); context anchor proA
  • Primary literature: none located yet; annotation rests on the domain/homology sources above.

Ancestral MTBC0 protein sequence

>mtbc0_002598|Rv2439c|proB
MRSPHRDAIRTARGLVVKVGTTALTTPSGMFDAGRLAGLAEAVERRMKAGSDVVIVSSGAIAAGIEPLGLSRRPKDLATKQAAASVGQVALVNSWSAAFARYGRTVGQVLLTAHDISMRVQHTNAQRTLDRLRALHAVAIVNENDTVATNEIRFGDNDRLSALVAHLVGADALVLLSDIDGLYDCDPRKTADATFIPEVSGPADLDGVVAGRSSHLGTGGMASKVSAALLAADAGVPVLLAPAADAATALADASVGTVFAARPARLSARRFWVRYAAEATGALTLDAGAVRAVVRQRRSLLAAGITAVSGRFCGGDVVELRAPDAAMVARGVVAYDASELATMVGRSTSELPGELRRPVVHADDLVAVSAKQAKQV