Rv3720 Resolved · high auto-curated

H37Rv Rv3720 · MTBC0 - · 420 aa · 4163736–4164998 (+) · RefSeq NP_218237.3

Annotation: from legacy to revised

Legacy (H37Rv / Mycobrowser)fatty acid synthase
MTBC0 PGAP re-annotation
Revised (this work)Fatty acid synthase. Pfam: CMAS (PF02353.27), Methyltransf_23 (PF13489.13), Methyltransf_25 (PF13649.13), Methyltransf_11 (PF08241.19).

Auto-curated: this verdict and function were generated by rules from PGAP + Pfam + Foldseek and have not been hand-reviewed.

Annotated on the H37Rv protein: this gene has no 1:1 ancestral MTBC0 anchor (PE/PPE, paralogue, IS element, or otherwise unanchored CDS).

Curated reference (UniProt)

UniProt O69687 SwissProt · reviewed · Evidence at protein level
UniProt nameProbable fatty acid methyltransferase Rv3720
EC (curated) EC 2.1.1.-
Curated functionMay be a S-adenosylmethionine-dependent methyltransferase involved in fatty acid metabolism.

Functional vocabulary (eggNOG-mapper, orthology transfer)

COG category M Cell wall / membrane / envelope biogenesis
Preferred namecfa
eggNOG descriptionCyclopropane-fatty-acyl-phospholipid synthase
Orthologous groupCOG2230
EC number EC 2.1.1.79
KEGG orthology K00574
Gene Ontology (8) GO:0005575, GO:0005618, GO:0005623, GO:0005886, GO:0016020, GO:0030312, GO:0044464, GO:0071944

Orthology-based transfer (eggNOG 5.0.2, diamond). EC/KO/GO/CAZy are computed annotations, not manual curation; cross-check against the primary literature before treating a specific reaction as established.

Conservation & selection (intra-MTBC, 145 209 strains)

pN/pS 0.264 · purifying
Polymorphic sites (≥ 0.1% of strains) 5 synonymous, 4 missense, 0 nonsense, 0 frameshift

pN/pS from segregating SNPs (singletons removed) normalised by possible sites. Low pN/pS = purifying selection (a strong signal that a "hypothetical" is a real, constrained gene). A high pN/pS is ambiguous: relaxed constraint or positive selection (drug resistance, antigenic variation) inflate it; e.g. rpoB/katG/pncA score high here for resistance, not loss of function. A clonal disruption (one allele over a clade) suggests lineage pseudogenisation; a convergent one (many independent alleles) is typical of resistance loss-of-function.

Domains (Pfam, hmmscan --cut_ga)

PfamAccessioni-EvalueResiduesDescription
CMASPF02353.27 9.4e-101130–398 Mycolic acid cyclopropane synthetase
Methyltransf_23PF13489.13 5.7e-10184–300 Methyltransferase domain
Methyltransf_25PF13649.13 8.4e-10196–289 Methyltransferase domain
Methyltransf_11PF08241.19 1.2e-09196–291 Methyltransferase domain

Functional interaction network (STRING v12, guilt-by-association)

PartnerProductScoreNo text-miningChannels (≥400)
Rv3719 hyp hypothetical protein 951 951 ctx neighborhood:859 cooccurence:648
Rv3718c hyp hypothetical protein 774 774 ctx neighborhood:773
Rv0448c hyp hypothetical protein 472 451
Rv0447c ufaA1 cyclopropane-fatty-acyl-phospholipid synthase UfaA 400 400
Rv3396c guaA GMP synthase 426 266
Rv3502c 3-oxoacyl-ACP reductase 429 72 textmining:411
Rv0242c fabG4 3-oxoacyl-ACP reductase FabG 454 71 textmining:437
Rv3254 hyp hypothetical protein 808 66 textmining:803
Rv3304 hyp hypothetical protein 639 66 textmining:630
Rv2488c LuxR family transcriptional regulator 422 63 textmining:409
Rv3253c cationic amino acid transport integral membrane protein 636 58 textmining:630
Rv0386 transcriptional regulator 417 54 textmining:409
Rv3302c glpD2 glycerol-3-phosphate dehydrogenase 514 50 textmining:510
Rv1589 bioB biotin synthetase 442 50 textmining:437
Rv0373c carbon monoxyde dehydrogenase large subunit 413 48 textmining:409

STRING combines evidence channels (neighborhood, fusion, cooccurrence, coexpression, experimental, database, text-mining) into a 0–1000 score. The ctx badge marks edges carried by the genomic-context channels (conserved neighborhood, fusion, phylogenetic co-occurrence), which are independent of orthology and structure and the strongest signal for an unknown gene. The no text-mining column recomputes the score from data alone, so a link that does not depend on the literature is visible. Association is a function hypothesis, not proof: corroborate with the operon context and the primary literature before assigning a function.

Evidence

  • Annotation from H37Rv (no MTBC0 1:1 anchor; H37Rv protein used): fatty acid synthase
  • Pfam (hmmscan --cut_ga): CMAS PF02353.27 (E=9e-101), Methyltransf_23 PF13489.13 (E=6e-10), Methyltransf_25 PF13649.13 (E=8e-10), Methyltransf_11 PF08241.19 (E=1e-09)
  • (auto-curated by rules from PGAP + Pfam + Foldseek; not hand-reviewed)

Sources

  • Ancestral sequence & coordinates: Harrison LB et al. (2024), An imputed ancestral reference genome for the MTBC, doi:10.1101/2023.09.07.556366
  • Product annotation: NCBI PGAP on MTBC0; legacy from H37Rv NC_000962.3 (RefSeq NP_218237.3)
  • Domains: Pfam-A via hmmscan --cut_ga — CMAS (PF02353.27), Methyltransf_23 (PF13489.13), Methyltransf_25 (PF13649.13), Methyltransf_11 (PF08241.19)
  • Sequence-level signal: ESM Atlas (EvolutionaryScale × BioHub) — exploratory
  • Controlled vocabulary: eggNOG-mapper 2.1.12 (Cantalapiedra et al. 2021, doi:10.1093/molbev/msab293), eggNOG 5.0 DB (Huerta-Cepas et al. 2019) — OG COG2230
  • Curated reference: UniProt O69687 (SwissProt, reviewed; Evidence at protein level)
  • Intra-MTBC selection: pN/pS and disruption from SPDI variants of 145 209 MTBC strains (this work, local collection vs H37Rv NC_000962.3)
  • Interaction network: STRING v12.0 (Szklarczyk et al. 2023, doi:10.1093/nar/gkac1000), taxon 83332, CC-BY 4.0 — 21 functional partner(s)
  • Primary literature: none located yet; annotation rests on the domain/homology sources above.

Ancestral MTBC0 protein sequence

>H37Rv|Rv3720|
MAEILEIFTATGQHPLKFTAYDGSTAGQDDATLGLDLRTPRGATYLATAPGELGLARAYVSGDLQAHGVHPGDPYELLKTLTERVDFKRPSARVLANVVRSIGVEHILPIAPPPQEARPRWRRMANGLLHSKTRDAEAIHHHYDVSNNFYEWVLGPSMTYTCAVFPNAEASLEQAQENKYRLIFEKLRLEPGDRLLDVGCGWGGMVRYAARRGVRVIGATLSAEQAKWGQKAVEDEGLSDLAQVRHSDYRDVAETGFDAVSSIGLTEHIGVKNYPFYFGFLKSKLRTGGLLLNHCITRHDNRSTSFAGGFTDRYVFPDGELTGSGRITTEIQQVGLEVLHEENFRHHYAMTLRDWCGNLVEHWDDAVAEVGLPTAKVWGLYMAASRVAFERNNLQLHHVLATKVDPRGDDSLPLRPWWQP